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Disease Info Card
Excessive Tearing
Information about Excessive Tearing: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Excessive Tearing
Most recent studies have shown that Excessive Tearing shares some biological mechanisms with cicatrix, conjunctival-diseases, conjunctival-hyperemia, conjunctivitis, corneal-diseases, dry-eye-syndromes, edema, fracture, headache, hemorrhage, inflammation, lacrimal-apparatus-diseases, lacrimal-duct-obstruction, neoplasms, pain, photophobia, pruritus, rhinorrhea, stenosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Excessive Tearing, and have been seen in publications frequently: Aging, Coagulation, Dehiscence, Delamination, Enucleation, Granuloma Formation, Hemostasis, Hypersensitivity, Inflammatory Response, Innervation, Localization, Pathogenesis, Reflex, Regeneration, Secretion, Tear Secretion, Translation, Transport, Transposition, Wound Healing
Quite a number of genes have been found to play important roles in Excessive Tearing, such as ACLY, CAT, CLIP1, CRAT, CSF2, DCXR, EGF, GLYAT, IL13, LAMC2, NLRP5, NR1I2, POMC, RANGAP1, SLC17A5. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.