Ceroid Lipofuscinosis, Neuronal, 5

Overview of Ceroid Lipofuscinosis, Neuronal, 5

Most recent studies have shown that Ceroid Lipofuscinosis, Neuronal, 5 shares some biological mechanisms with ataxia, atrophy, blind-vision, brain-diseases, cerebral-atrophy, clumsiness, dysequilibrium-syndrome, encephalopathies, epilepsy, infantile-neuronal-ceroid-lipofuscinosis, late-infantile-neuronal-ceroid-lipfuscinosis, lysosomal-storage-diseases, nerve-degeneration, nervousness, neurodegenerative-disorders, neuronal-ceroid-lipofuscinoses.

Among the many pathways, these few ones have gauged particular interests from scientists studying Ceroid Lipofuscinosis, Neuronal, 5, and have been seen in publications frequently: Aging, Cell Death, Endocytosis, Immune Response, Inflammatory Response, Interphase, Localization, Metaphase, Pathogenesis, Transport

Quite a number of genes have been found to play important roles in Ceroid Lipofuscinosis, Neuronal, 5, such as ACD, BTF3, BTN3A2, CLN3, CLN5, CLN6, CLN8, DHX9, DNAJC5, ITGB3, JUN, LAMP3, MFSD8, NCL, PPT1, TPP1, TTPA, VCAN. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Ceroid Lipofuscinosis, Neuronal, 5 Related Genes

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Pathways Related to Ceroid Lipofuscinosis, Neuronal, 5

This information is being compiled and will come in a future update

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