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Disease Info Card
Central Nervous System Sensitization
Information about Central Nervous System Sensitization: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Central Nervous System Sensitization
Most recent studies have shown that Central Nervous System Sensitization shares some biological mechanisms with acute-onset-pain, chronic-pain, depressive-disorder, fibromyalgia, headache, hyperalgesia, hyperalgesias-mechanical, inflammation, migraine-disorders, nerve-damage, nervousness, neuralgia, pain, pain-postoperative, pain-syndrome, peripheral-nerve-injuries, peripheral-neuropathy, thermal-hyperalgesia.
Among the many pathways, these few ones have gauged particular interests from scientists studying Central Nervous System Sensitization, and have been seen in publications frequently: Cell Activation, Cytokine Production, Habituation, Hypersensitivity, Inflammatory Response, Innervation, Interphase, Intracellular Signal Transduction, Localization, Locomotion, Micturition, Muscle Contraction, Pathogenesis, Reflex, Response To Pain, Secretion, Sensitization, Sensory Processing, Synaptic Transmission, Vasodilation
Quite a number of genes have been found to play important roles in Central Nervous System Sensitization, such as AURKA, BDNF, CALCA, EPHB2, FOS, GRIN1, IL6, MAPK1, NDOR1, PPP5C, PPT1, PTGS2, RPL5, S100A12, TAC1, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.