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Disease Info Card
Brain Damage, Chronic
Information about Brain Damage, Chronic: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Brain Damage, Chronic
Most recent studies have shown that Brain Damage, Chronic shares some biological mechanisms with aphasia, brain-diseases, brain-injuries, brain-ischemia, cerebral-hemorrhage, cerebral-infarction, cerebrovascular-accident, cognition-disorders, comatose, craniocerebral-trauma, encephalopathies, epilepsy, hemorrhage, learning-disorders, malnutrition, mental-disorders, schizophrenia, traumatic-brain-injury.
Among the many pathways, these few ones have gauged particular interests from scientists studying Brain Damage, Chronic, and have been seen in publications frequently: Aging, Brain Development, Cell Death, Coagulation, Cognition, Excretion, Habituation, Inflammatory Response, Localization, Long-term Memory, Myelination, Neurogenesis, Neuroprotection, Pathogenesis, Reflex, Secretion, Short-term Memory, Social Behavior, Transport, Visual Perception
Quite a number of genes have been found to play important roles in Brain Damage, Chronic, such as ALB, CA1, CA3, CAT, CRAT, CSF2, ENO1, ENO2, FDFT1, GFAP, GLYAT, LAMC2, MMEL1, PLXNB1, POMC, RHD, S100B, SHOX, TNFSF10. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.