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- Table of Contents
Information about Benign Recurrent Intrahepatic Cholestasis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Benign Recurrent Intrahepatic Cholestasis shares some biological mechanisms with biliary-cirrhosis, cholangitis, cholangitis-sclerosing, cholelithiasis, cholestasis, cholestasis-of-pregnancy, fibrosis, hepatitis, hyperbilirubinemia, icterus, intrahepatic-cholestasis, jaundice-obstructive, liver-cirrhosis, liver-damage, liver-diseases, liver-failure, pregnancy-complications, primary-biliary-cirrhosis, progressive-intrahepatic-cholestasis-(disorder), pruritus.
Among the many pathways, these few ones have gauged particular interests from scientists studying Benign Recurrent Intrahepatic Cholestasis, and have been seen in publications frequently: Acid Secretion, Aminophospholipid Transport, Bile Acid Secretion, Cellular Localization, Complement Activation, Conjugation, Excretion, Gallstone Formation, Localization, Pathogenesis, Reflex, Secretion, Transport
Quite a number of genes have been found to play important roles in Benign Recurrent Intrahepatic Cholestasis, such as ABCB11, ABCB4, ABCC2, ALB, ATP8A1, ATP8B1, C3, DLAT, FABP6, GGT1, GGT2, GGTLC1, ITGA2B, ITGB3, JAG1, NR1H4, SLA2, TMEM30A. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.