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Information about Acyl-coa Dehydrogenase, Very Long-chain Deficiency: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Acyl-coa Dehydrogenase, Very Long-chain Deficiency shares some biological mechanisms with cardiomyopathies, hepatomegaly, hypertrophic-cardiomyopathy, hypertrophy, hypoglycemia, inborn-errors-of-metabolism, long-chain-acyl-coa-dehydrogenase-deficiency, medium-chain-acyl-coenzyme-a-dehydrogenase-deficiency, metabolic-diseases, mitochondrial-diseases, muscle-hypotonia, myalgia, myopathy, pain, rhabdomyolysis, sudden-death, sudden-infant-death-syndrome, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Acyl-coa Dehydrogenase, Very Long-chain Deficiency, and have been seen in publications frequently: Aging, Carnitine Shuttle, Fatty Acid Beta-oxidation, Fatty Acid Oxidation, Gluconeogenesis, Glycolysis, Lipid Storage, Localization, Menopause, Oxidative Phosphorylation, Pathogenesis
Quite a number of genes have been found to play important roles in Acyl-coa Dehydrogenase, Very Long-chain Deficiency, such as ACADL, ACADM, ACADS, ACADVL, BRCA2, CALD1, CDH15, CHKA, CHKB, CPT2, FANCD2, MCAT, MCPH1, NBN, PPARG, SLC16A1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.
Aging | Carnitine Shuttle | Fatty Acid Beta oxidation |
Fatty Acid Oxidation | Gluconeogenesis | Glycolysis |
Lipid Storage | Localization | Menopause |
Oxidative Phosphorylation | Pathogenesis |