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Disease Info Card
Sudden Infant Death Syndrome
Information about Sudden Infant Death Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Sudden Infant Death Syndrome
Most recent studies have shown that Sudden Infant Death Syndrome shares some biological mechanisms with airway-obstruction, anoxia, apnea, apparent-life-threatening-event-in-infant, asphyxia, bradycardia, cardiac-arrhythmia, congenital-abnormality, gastroesophageal-reflux-disease, heart-diseases, hemorrhage, hypoxia, infective-disorder, long-qt-syndrome, nervousness, pregnancy-complications, respiratory-tract-infections, sleep-apnea-syndromes, sudden-death.
Among the many pathways, these few ones have gauged particular interests from scientists studying Sudden Infant Death Syndrome, and have been seen in publications frequently: Aging, Anaphylaxis, Brain Development, Cardiac Conduction, Cell Death, Excretion, Fatty Acid Oxidation, Hypersensitivity, Immune Response, Inflammatory Response, Innervation, Lactation, Localization, Myelination, Pathogenesis, Reflex, Response To Hypoxia, Secretion, Transport, Vasoconstriction
Quite a number of genes have been found to play important roles in Sudden Infant Death Syndrome, such as ACADM, ALB, C2, CDH15, CP, CSF2, EXOSC10, GFAP, IL6, LAMC2, POMC, POR, SCN5A, SLC6A4, TAC1, TEAD1, TH, TNF, ZBED1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.