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- Table of Contents
, No Gclids
pathway Info Card
Mediolateral Intercalation
Information about Mediolateral Intercalation: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Mediolateral Intercalation
Most recent studies have shown that Mediolateral Intercalation shares some biological mechanisms with cell-adhesion, cell-division, cell-motility, contact-inhibition, convergent-extension, epiboly, gastrulation, localization, microtubule-depolymerization, muscle-contraction, notochord-morphogenesis, segmentation, tail-morphogenesis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Mediolateral Intercalation, and have been seen in publications frequently: cell-adhesion, cell-division, cell-motility, contact-inhibition, convergent-extension, epiboly, gastrulation, localization, microtubule-depolymerization, muscle-contraction, notochord-morphogenesis, segmentation, tail-morphogenesis
Quite a number of genes have been found to play important roles in Mediolateral Intercalation, such as ADAR, Arc, BMP1, CDH1, CHM, MIB1, MPZ, NOL3, PGPEP1, PIEZO1, PRCP, SHH, SPON1, TFF2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this pathway. Plesae stay updated.
Diseases Related to Mediolateral Intercalation
This information is being compiled and will come in a future update
| morphologically altered structure | nervousness | overlying behavior |
| tissue adhesions |