Tyrosine Transaminase Deficiency Disease

Overview of Tyrosine Transaminase Deficiency Disease

Most recent studies have shown that Tyrosine Transaminase Deficiency Disease shares some biological mechanisms with corneal-diseases, dermatologic-disorders, disorder-of-eye, dystrophy, hereditary-corneal-dystrophy, hyperkeratosis, hypertyrosinemia, inborn-errors-of-metabolism, keratitis, keratitis-dendritic, keratosis, metabolic-diseases, palmoplantar-keratosis, phenylketonurias, photophobia, tyrosinemias, ulcer.

Among the many pathways, these few ones have gauged particular interests from scientists studying Tyrosine Transaminase Deficiency Disease, and have been seen in publications frequently: Acute Inflammatory Response, Brain Development, Excretion, Gluconeogenesis, Inflammatory Response, Lipid Storage, Localization, Metaphase, Pathogenesis, Protein Oxidation

Quite a number of genes have been found to play important roles in Tyrosine Transaminase Deficiency Disease, such as ATAT1, CAT, CHKA, CHKB, CHST6, CRAT, G6PD, GLYAT, KCNE1, KRT12, KRT3, MINK1, QPCT, TACSTD2, TAT, TGFBI, TNFSF14, TP63, TYR. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Tyrosine Transaminase Deficiency Disease Related Genes

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Pathways Related to Tyrosine Transaminase Deficiency Disease

This information is being compiled and will come in a future update

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