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Disease Info Card
Turcot Syndrome (disorder)
Information about Turcot Syndrome (disorder): characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Turcot Syndrome (disorder)
Most recent studies have shown that Turcot Syndrome (disorder) shares some biological mechanisms with adenocarcinoma, adenoma, adenomatous-polyposis-coli, brain-neoplasms, carcinogenesis, carcinoma, cell-transformation-neoplastic, colonic-neoplasms, colorectal-cancer, colorectal-neoplasms, glioblastoma, glioma, hereditary-nonpolyposis-colorectal-cancer, malignant-neoplasms, malignant-paraganglionic-neoplasm, malignant-tumor-of-colon, microsatellite-instability, neoplasms, polyposis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Turcot Syndrome (disorder), and have been seen in publications frequently: Aging, Cell Cycle, Cell Cycle Arrest, Cell Cycle Checkpoint, Cell Death, Cell Killing, Cell Proliferation, Dna Methylation, Dna Repair, Dna Replication, Drug Resistance, Gene Silencing, Hypersensitivity, Localization, Methylation, Mismatch Repair, Oncogenesis, Pathogenesis, Regeneration, Translation
Quite a number of genes have been found to play important roles in Turcot Syndrome (disorder), such as APC, BAX, BRAF, CEL, FAP, GLMN, KRAS, MLH1, MRC1, MSH2, MSH3, MSH6, PMS2, PROC, PTEN, SIL1, TP53. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.