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Disease Info Card
Truncus Arteriosus, Persistent
Information about Truncus Arteriosus, Persistent: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Truncus Arteriosus, Persistent
Most recent studies have shown that Truncus Arteriosus, Persistent shares some biological mechanisms with atresia, congenital-abnormality, congenital-atresia-of-pulmonary-valve, congenital-heart-defects, congenital-heart-disease, crest-syndrome, digeorge-syndrome, double-outlet-right-ventricle, heart-diseases, heart-failure, heart-septal-defects, interrupted-aortic-arch, patent-ductus-arteriosus, pulmonary-stenosis, regurgitation, stenosis, tetralogy-of-fallot, transposition-of-great-vessels, ventricular-septal-defects.
Among the many pathways, these few ones have gauged particular interests from scientists studying Truncus Arteriosus, Persistent, and have been seen in publications frequently: Angiogenesis, Artery Development, Cell Death, Cell Differentiation, Cell Migration, Cell Proliferation, Coagulation, Developmental Process, Gastrulation, Hatching, Heart Development, Innervation, Localization, Neural Crest Cell Migration, Outflow Tract Morphogenesis, Pathogenesis, Somite Specification, Transport, Transposition, Vasculogenesis
Quite a number of genes have been found to play important roles in Truncus Arteriosus, Persistent, such as AMY2A, APP, ATP6V0A1, ATP6V0A4, BLOC1S6, ELN, EPB42, F11, GDF1, NKX2-5, PLP2, PRH1, PTCRA, RFC1, RFC2, RFC4, SGCG, SS18L1, TBX1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.