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Disease Info Card
Total Colonic Aganglionosis
Information about Total Colonic Aganglionosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Total Colonic Aganglionosis
Most recent studies have shown that Total Colonic Aganglionosis shares some biological mechanisms with anastomotic-leak, atresia, constipation, crest-syndrome, down-syndrome, dysplasia, enterocolitis, enterostomy-(morphologic-abnormality), fecal-incontinence, hirschsprung-disease, hypoventilation, intestinal-atresia, intestinal-obstruction, megacolon, multiple-endocrine-neoplasia-type-2a, nervousness, pathological-dilatation, stenosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Total Colonic Aganglionosis, and have been seen in publications frequently: Cell Adhesion, Cell Development, Cell Growth, Cell Migration, Cell Proliferation, Defecation, Dehiscence, Enteric Nervous System Development, Innervation, Localization, Mrna Splicing, Nervous System Development, Neural Crest Cell Development, Neural Crest Cell Migration, Neuroblast Migration, Pathogenesis, Peristalsis, Reflex, System Development, Transport
Quite a number of genes have been found to play important roles in Total Colonic Aganglionosis, such as ACHE, CCDC6, EDN3, EDNRB, ELL, ENO1, ENO2, GDNF, HTT, INPP5K, KIT, NRTN, PHOX2B, PRPH, RET, SOX10, SS18L1, TAPBP, UCHL1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.