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Disease Info Card
Enterostomy (morphologic Abnormality)
Information about Enterostomy (morphologic Abnormality): characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Enterostomy (morphologic Abnormality)
Most recent studies have shown that Enterostomy (morphologic Abnormality) shares some biological mechanisms with atresia, carcinoma-neuroendocrine, crohn-disease, enterocolitis, enterocolitis-necrotizing, enterocolitis-pseudomembranous, hemorrhage, hernia, hirschsprung-disease, ileus, intestinal-diseases, intestinal-obstruction, intestinal-perforation, malignant-neoplasms, pathologic-fistula, pathological-dilatation, peritonitis, stenosis, systemic-infection.
Among the many pathways, these few ones have gauged particular interests from scientists studying Enterostomy (morphologic Abnormality), and have been seen in publications frequently: Aging, Defecation, Dehiscence, Eating Behavior, Excretion, Gastric Emptying, Gastric Motility, Hemostasis, Inflammatory Response, Innervation, Intestinal Absorption, Localization, Pathogenesis, Peristalsis, Secretion, Translation, Transport, Transposition, Virulence, Wound Healing
Quite a number of genes have been found to play important roles in Enterostomy (morphologic Abnormality), such as ALB, CAT, CD55, CFTR, CRAT, CYLD, GLUL, GLYAT, GNAI1, HPN, IL6, NT5E, OPN1SW, SLPI, SMOC1, SMYD1, TAPBP, TNF, USB1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.