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Disease Info Card
Tinea Cruris
Information about Tinea Cruris: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Tinea Cruris
Most recent studies have shown that Tinea Cruris shares some biological mechanisms with candidiasis, cutaneous-candidiasis, dermatitis, dermatologic-disorders, dermatomycoses, infective-disorder, intertrigo, mycoses, onychomycosis, pruritus, tinea, tinea-barbae, tinea-capitis, tinea-corporis-(disorder), tinea-faciei, tinea-manuum, tinea-pedis, tinea-versicolor.
Among the many pathways, these few ones have gauged particular interests from scientists studying Tinea Cruris, and have been seen in publications frequently: Aging, Blood Circulation, Colony Morphology, Fermentation, Germination, Hypersensitivity, Localization, Pathogenesis, Regeneration, Sensitization, Wound Healing
Quite a number of genes have been found to play important roles in Tinea Cruris, such as AKR1B1, ALB, BCAR1, CBS, CSE1L, CTNND1, EMD, EPO, FLG, GLYAT, GSTM4, GSTT1, ITCH, OTC, TDO2, TMEFF2, TXNRD2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.
Pathways Related to Tinea Cruris
This information is being compiled and will come in a future update
| Aging | Blood Circulation | Colony Morphology |
| Fermentation | Germination | Hypersensitivity |
| Localization | Pathogenesis | Regeneration |
| Sensitization | Wound Healing |