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- Table of Contents
Information about T(2;5)(p23;q35): characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that T(2;5)(p23;q35) shares some biological mechanisms with anaplastic-lymphoma, b-cell-lymphomas, cell-transformation-neoplastic, chromosomal-translocation, cytogenetic-abnormality, diffuse-large-b-cell-lymphoma, hodgkin-disease, ki-1--anaplastic-large-cell-lymphoma, leukemia, lymphoma, lymphoma-non-hodgkin, lymphomatoid-papulosis, lymphoproliferative-disorders, malignant-neoplasms, malignant-paraganglionic-neoplasm, neoplasms, skin-neoplasms, t-cell-lymphoma, trisomy.
Among the many pathways, these few ones have gauged particular interests from scientists studying T(2;5)(p23;q35), and have been seen in publications frequently: Cell Adhesion, Cell Cycle, Cell Cycle Arrest, Cell Death, Cell Growth, Cell Proliferation, Glycosylation, Hyperphosphorylation, Induction Of Apoptosis, Interphase, Localization, Metaphase, Oncogenesis, Pathogenesis, Phagocytosis, Programmed Cell Death, Reverse Transcription, Rna Interference, Translation, Transport
Quite a number of genes have been found to play important roles in T(2;5)(p23;q35), such as ACVRL1, AKT1, ALK, CDK5R1, CDKN1A, COIL, ETFA, HLA-DQA1, IL1R1, MUC1, NOD2, NPM1, PTGES3, RRAS, SLPI, TMED10, TNFRSF8, TPT1, WDR48. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.