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Disease Info Card
Stinging Sensation
Information about Stinging Sensation: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Stinging Sensation
Most recent studies have shown that Stinging Sensation shares some biological mechanisms with allergy, anaphylaxis, contact-dermatitis, dermatitis, dermatologic-disorders, edema, erythema, glaucoma, glaucoma-open-angle, hypertensive-disease, immediate-hypersensitivity, inflammation, intraocular-pressure-disorder, ocular-hypertension, pain, pruritus, sting-injury, urticaria.
Among the many pathways, these few ones have gauged particular interests from scientists studying Stinging Sensation, and have been seen in publications frequently: Anaphylaxis, Cell Proliferation, Coagulation, Cytokine Production, Excretion, Exocytosis, Hypersensitivity, Inflammatory Response, Localization, Locomotion, Mast Cell Degranulation, Pathogenesis, Pigmentation, Reflex, Secretion, Sensitization, Swimming, Transport, Vasodilation, Wound Healing
Quite a number of genes have been found to play important roles in Stinging Sensation, such as C2, CALM1, CALM2, CALM3, CAMKMT, CAT, FECH, IL13, ITCH, KRIT1, NOD2, PLA2G1B, PLB1, SHBG, TAC1, TNF, WAS, YWHAZ. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.