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Disease Info Card
Sting Injury
Information about Sting Injury: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Sting Injury
Most recent studies have shown that Sting Injury shares some biological mechanisms with allergy, anaphylaxis, arachnidism, asthma, edema, immediate-hypersensitivity, pain, poisoning, pulmonary-edema, snake-bites, stinging-sensation, urticaria.
Among the many pathways, these few ones have gauged particular interests from scientists studying Sting Injury, and have been seen in publications frequently: Anaphylaxis, Basophil Activation, Cell Activation, Coagulation, Cytokine Production, Hypersensitivity, Immune Response, Inflammatory Response, Innate Immune Response, Localization, Mast Cell Activation, Mast Cell Degranulation, Pathogenesis, Reflex, Secretion, Sensitization, Swimming, Synaptic Transmission, Transport, Vasoconstriction
Quite a number of genes have been found to play important roles in Sting Injury, such as CD63, IGHE, IL10, IL13, IL4, IL6, IRF3, PLA2G1B, PLB1, SCTR, SNCG, TBK1, TNF, VIT, WAS, YWHAZ. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.