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Disease Info Card
Serous Retinal Detachment
Information about Serous Retinal Detachment: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Serous Retinal Detachment
Most recent studies have shown that Serous Retinal Detachment shares some biological mechanisms with blind-vision, central-serous-chorioretinopathy, chorioretinal-disorder, choroid-diseases, choroid-neoplasms, edema, glaucoma, hemangioma, hemorrhage, inflammation, macular-retinal-edema, macule, neoplasms, pathologic-neovascularization, retinal-detachment, retinal-diseases, uveitis, uveomeningoencephalitic-syndrome, visual-impairment.
Among the many pathways, these few ones have gauged particular interests from scientists studying Serous Retinal Detachment, and have been seen in publications frequently: Aging, Cell Activation, Coagulation, Complement Activation, Enucleation, Fibroblast Proliferation, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Ossification, Pathogenesis, Pigmentation, Reflex, Regeneration, Segmentation, Sensitization, Transport, Vasculogenesis, Vasoconstriction
Quite a number of genes have been found to play important roles in Serous Retinal Detachment, such as BEST1, CAT, COX5A, CRAT, ERG, FANCA, FXN, GLYAT, HLA-DRB4, KCNH2, MID1, OCA2, PEA15, PLXNA2, RPE, SLC2A1, TNFSF14, VEGFA, VHL. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.