Disease Info Card

Secondary Hyperaldosteronism

Information about Secondary Hyperaldosteronism: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Secondary Hyperaldosteronism

Most recent studies have shown that Secondary Hyperaldosteronism shares some biological mechanisms with alkalosis, bartter-disease, conn-syndrome, edema, essential-hypertension, fibrosis, heart-failure, hyperaldosteronism, hyperplasia, hypertension-renovascular, hypertensive-disease, hypertrophy, kidney-diseases, kidney-neoplasm, liver-cirrhosis, malignant-hypertension, neoplasms, renal-artery-obstruction, renal-hypertension, stenosis.

Among the many pathways, these few ones have gauged particular interests from scientists studying Secondary Hyperaldosteronism, and have been seen in publications frequently: Aging, Aldosterone Secretion, Chloride Transport, Diuresis, Electron Transport, Excretion, Flight, Glomerular Filtration, Hormone Secretion, Ion Transport, Natriuresis, Pathogenesis, Prolactin Secretion, Reflex, Regulation Of Aldosterone Secretion, Secretion, Transport, Transposition, Vasoconstriction, Vasodilation

Quite a number of genes have been found to play important roles in Secondary Hyperaldosteronism, such as ACE, AGT, ALB, AMY2A, ANG, AVP, BLOC1S6, CYP11B2, EPB42, NR3C2, POMC, PRH1, PRL, PTH, PTRH1, REN, S100A6, SLC12A3. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.