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Disease Info Card
Retinal Drusen
Information about Retinal Drusen: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Retinal Drusen
Most recent studies have shown that Retinal Drusen shares some biological mechanisms with age-related-macular-degeneration, atrophy, basal-laminar-drusen-(disorder), blind-vision, choroidal-neovascularization, disorder-of-eye, dystrophy, exudative-age-related-macular-degeneration, geographic-atrophy, glomerulonephritis-membranoproliferative, hard-drusen, hyperpigmentation, macule, maculopathy, pathologic-neovascularization, retinal-detachment, retinal-diseases, soft-drusen, visual-impairment.
Among the many pathways, these few ones have gauged particular interests from scientists studying Retinal Drusen, and have been seen in publications frequently: Aging, Angiogenesis, Autophagy, Cell Migration, Complement Activation, Dna Repair, Glycosylation, Inflammatory Response, Localization, Neurogenesis, Pathogenesis, Pigmentation, Protein Secretion, Proteolysis, Reflex, Secretion, Segmentation, Tissue Development, Transport, Water Transport
Quite a number of genes have been found to play important roles in Retinal Drusen, such as ABCA4, AGA, AMD1, APOE, BEST1, C3, CFB, CFH, CLU, EFEMP1, FANCA, FXN, GLS2, MID1, PLXNA2, RPE, TIMP3, VEGFA, VTN. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.