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Disease Info Card
Pterygium Colli
Information about Pterygium Colli: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Pterygium Colli
Most recent studies have shown that Pterygium Colli shares some biological mechanisms with acquired-cubitus-valgus, blepharoptosis, congenital-abnormality, congenital-heart-defects, congenital-webbing, cryptorchidism, cytogenetic-abnormality, dwarfism, dysplasia, embryonic-mosaic, low-set-ears, lymphedema, microcephaly, micrognathism, noonan-syndrome, orbital-separation-excessive, pterygium, trisomy, turner-syndrome.
Among the many pathways, these few ones have gauged particular interests from scientists studying Pterygium Colli, and have been seen in publications frequently: Anagen, Anaphase, Chromosome Breakage, Coagulation, Development Of Secondary Sexual Characteristics, Fatty Acid Transport, Keratinization, Localization, Meiosis, Meiosis Ii, Menarche, Menstruation, Mitosis, Ossification, Pathogenesis, Pigmentation, Segmentation, Sister Chromatid Segregation, Transport, Transposition
Quite a number of genes have been found to play important roles in Pterygium Colli, such as AFP, BRD2, CACNA1C, CDK5R1, ENDOU, EXOSC6, INVS, KRAS, LCN2, MAP2K1, PTPN11, S100A10, TBXAS1, TMED9, TPPP, TYMS. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.