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Disease Info Card
Primary Hypogonadism
Information about Primary Hypogonadism: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Primary Hypogonadism
Most recent studies have shown that Primary Hypogonadism shares some biological mechanisms with amenorrhea, atrophy, congenital-abnormality, delayed-puberty, diabetes-mellitus, endocrine-system-diseases, erectile-dysfunction, gonadal-dysgenesis, gynecomastia, hypogonadism, hypogonadotropic-hypogonadism, infertility, klinefelter-syndrome, neoplasms, pituitary-diseases, premature-menopause, primary-physiologic-amenorrhea, testicular-hypogonadism, turner-syndrome.
Among the many pathways, these few ones have gauged particular interests from scientists studying Primary Hypogonadism, and have been seen in publications frequently: Aging, Cell Maturation, Excretion, Fertilization, Glycosylation, Gonadotropin Secretion, Hormone Secretion, Luteinizing Hormone Secretion, Menarche, Menopause, Menstruation, Muscle Atrophy, Ovulation, Pathogenesis, Response To Gonadotropin-releasing Hormone, Secretion, Sex Determination, Spermatogenesis, Translation, Transport
Quite a number of genes have been found to play important roles in Primary Hypogonadism, such as BRD2, CYP17A1, CYP19A1, DBT, DMD, GGH, GH1, GNRH1, IGF1, INS, LHCGR, PBX1, PLOD1, POMC, PRL, SHBG, SRY, TRH. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.