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Disease Info Card
Plaque Psoriasis
Information about Plaque Psoriasis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Plaque Psoriasis
Most recent studies have shown that Plaque Psoriasis shares some biological mechanisms with arthritis, arthritis-psoriatic, autoimmune-reaction, dental-plaque, dermatitis, dermatitis-atopic, dermatologic-disorders, erythema, guttate-psoriasis, infective-disorder, inflammation, malignant-neoplasms, pain, polyendocrinopathies-autoimmune, pruritus, psoriasis, pustular-psoriasis, rheumatoid-arthritis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Plaque Psoriasis, and have been seen in publications frequently: Angiogenesis, Cell Activation, Cell Adhesion, Cell Differentiation, Cell Proliferation, Cytokine Production, Excretion, Hypersensitivity, Immune Response, Induction Of Apoptosis, Inflammatory Response, Keratinization, Keratinocyte Differentiation, Keratinocyte Proliferation, Localization, Pathogenesis, Pigmentation, Secretion, T Cell Activation, Transport
Quite a number of genes have been found to play important roles in Plaque Psoriasis, such as ARHGEF2, CD2, CD4, EBNA1BP2, HLA-C, IL2, IL23A, IL37, IL9, ITGAL, KRT10, LANCL1, MAPK1, PSMD7, PTH, RABEPK, RPSA, TNF, TP63, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.