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Disease Info Card
Placental Insufficiency
Information about Placental Insufficiency: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Placental Insufficiency
Most recent studies have shown that Placental Insufficiency shares some biological mechanisms with congenital-abnormality, diabetes-mellitus, fetal-death, fetal-diseases, fetal-distress, fetal-growth-retardation, fetal-hypoxia, growth-retardation, high-risk-pregnancy, hypertensive-disease, hypoxia, malnutrition, placenta-disorders, pre-eclampsia, pregnancy-complications, pregnancy-complications-cardiovascular, pregnancy-in-diabetics, premature-birth, premature-obstetric-labor.
Among the many pathways, these few ones have gauged particular interests from scientists studying Placental Insufficiency, and have been seen in publications frequently: Angiogenesis, Blood Circulation, Brain Development, Coagulation, Complement Activation, Excretion, Fertilization, Glomerular Filtration, Hemostasis, Insulin Secretion, Lactation, Lung Development, Mating, Myelination, Parturition, Pathogenesis, Platelet Activation, Secretion, Transport, Vasoconstriction
Quite a number of genes have been found to play important roles in Placental Insufficiency, such as AFP, AGA, AGT, BDNF, CSH1, CSH2, FGR, IGF1, IGF2, INS, LEP, LGALS1, LNPEP, NR3C1, PLXNA2, POMC, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.