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Disease Info Card
Periodontal Attachment Loss
Information about Periodontal Attachment Loss: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Periodontal Attachment Loss
Most recent studies have shown that Periodontal Attachment Loss shares some biological mechanisms with alveolar-bone-loss, chronic-periodontitis, crest-syndrome, dental-caries, dental-plaque, diabetes-mellitus, diabetes-mellitus-non-insulin-dependent, furcation-defects, gingival-recession, gingivitis, hemorrhage, infective-disorder, inflammation, osteopenia, periodontal-diseases, periodontal-pocket, periodontitis, periodontitis-juvenile, tooth-loss.
Among the many pathways, these few ones have gauged particular interests from scientists studying Periodontal Attachment Loss, and have been seen in publications frequently: Acute-phase Response, Aging, Angiogenesis, Bone Remodeling, Bone Resorption, Chemotaxis, Cytokine Production, Dehiscence, Glomerular Filtration, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Menopause, Pathogenesis, Regeneration, Secretion, Tissue Regeneration, Virulence, Wound Healing
Quite a number of genes have been found to play important roles in Periodontal Attachment Loss, such as CRP, EMD, EYA1, FBLIM1, GCFC2, GNAI1, GOPC, HPD, IL6, MMP8, OPN1SW, PAM, SHCBP1, SMYD1, SS18L1, TNF, UCN2, VSX1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.