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Disease Info Card
Pancreatic Injury
Information about Pancreatic Injury: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Pancreatic Injury
Most recent studies have shown that Pancreatic Injury shares some biological mechanisms with abscess, acute-pancreatitis, edema, fibrosis, hemorrhage, inflammation, multiple-trauma, nonpenetrating-wounds, pain, pancreatic-diseases, pancreatic-fistula, pancreatic-pseudocyst, pancreatitis, pancreatitis-acute-necrotizing, pancreatitis-alcoholic, pancreatitis-chronic, pathologic-fistula, trauma-to-the-abdomen, wounds-penetrating.
Among the many pathways, these few ones have gauged particular interests from scientists studying Pancreatic Injury, and have been seen in publications frequently: Acute Inflammatory Response, Autophagy, Cell Activation, Cell Death, Cell Differentiation, Cell Migration, Cell Proliferation, Chemokine Production, Cytokine Production, Excretion, Hemostasis, Immune Response, Inflammatory Response, Localization, Pathogenesis, Regeneration, Reverse Transcription, Secretion, Transdifferentiation, Trypsinogen Activation
Quite a number of genes have been found to play important roles in Pancreatic Injury, such as AMY2A, CCK, CCL2, CTSB, IL10, IL6, INS, ISYNA1, JUN, MPO, NFKB1, NOS2, RIPK2, SPINK1, SST, TLR4, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.