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Disease Info Card
Pancreatitis, Acute Necrotizing
Information about Pancreatitis, Acute Necrotizing: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Pancreatitis, Acute Necrotizing
Most recent studies have shown that Pancreatitis, Acute Necrotizing shares some biological mechanisms with abdominal-pain, abscess, acute-pancreatitis, bacterial-infections, cholecystolithiasis, edema, hemorrhage, infective-disorder, inflammation, inflammatory-response, multiple-organ-failure, necrosis-of-pancreas, pain, pancreatic-pseudocyst, pancreatitis, pancreatitis-chronic, pathologic-fistula, systemic-infection.
Among the many pathways, these few ones have gauged particular interests from scientists studying Pancreatitis, Acute Necrotizing, and have been seen in publications frequently: Autophagy, Cell Adhesion, Cell Cycle, Cell Death, Coagulation, Cytokine Production, Dehiscence, Excretion, Hemostasis, Immune Response, Induction Of Apoptosis, Inflammatory Response, Leukocyte Activation, Localization, Pathogenesis, Regeneration, Reverse Transcription, Secretion, Transport, Trypsinogen Activation
Quite a number of genes have been found to play important roles in Pancreatitis, Acute Necrotizing, such as ALB, APCS, CCK, CRP, CSRP1, IL10, IL6, MPO, NFKB1, NOS2, NPPA, PLA2G1B, PLB1, RANGAP1, SH2D1A, SLC17A5, TNF, YWHAZ. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.