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- Table of Contents
Information about Pancreatic Acinar Cell Carcinoma: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Pancreatic Acinar Cell Carcinoma shares some biological mechanisms with abdominal-pain, acinar-cell-carcinoma, adenocarcinoma, carcinoma, endocrine-gland-neoplasms, fat-necrosis, liver-carcinoma, liver-neoplasms, malignant-neoplasm-of-pancreas, malignant-neoplasms, malignant-paraganglionic-neoplasm, metastatic-malignant-neoplasm-to-the-liver, neoplasm-metastasis, neoplasms, pain, pancreatic-adenocarcinoma, pancreatic-neoplasm, pancreatitis, panniculitis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Pancreatic Acinar Cell Carcinoma, and have been seen in publications frequently: Acinar Cell Differentiation, Cell Development, Cell Differentiation, Cell Growth, Cell Proliferation, Cell-cell Adhesion, Dna Repair, Fucosylation, Intracellular Protein Transport, Invasive Growth, Localization, Methylation, Mismatch Repair, Mitosis, Pathogenesis, Protein Transport, Regulation Of Cell-cell Adhesion, Secretion, Transport
Quite a number of genes have been found to play important roles in Pancreatic Acinar Cell Carcinoma, such as ACACA, AFP, APC, CCK, CDH1, CLDN5, CLDN7, CTNNB1, EGFR, FUT8, INS, PLA2G1B, PROC, SMAD4, STK11, SYP, TP53, TRIM26. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.