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Disease Info Card
Panniculitis
Information about Panniculitis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Panniculitis
Most recent studies have shown that Panniculitis shares some biological mechanisms with dermatologic-disorders, erythema, fat-necrosis, inflammation, lupus-erythematosus-systemic, lymphoma, lymphoma-t-cell-cutaneous, mesenteric-panniculitis, neoplasms, nodular-panniculitis, pain, panniculitis-lobular, panniculitis-lupus-erythematosus, panniculitis-nodular-nonsuppurative, panniculitis-peritoneal, skin-neoplasms, t-cell-lymphoma, ulcer, vasculitis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Panniculitis, and have been seen in publications frequently: Angiogenesis, Cell Proliferation, Coagulation, Complement Activation, Cytokine Production, Fibrinolysis, Granuloma Formation, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Macrophage Activation, Muscle Atrophy, Pathogenesis, Phagocytosis, Pigmentation, Response To Tetracycline, Secretion, Sensitization, Wound Healing
Quite a number of genes have been found to play important roles in Panniculitis, such as C3, CAT, CD4, CD8A, CRAT, CTLA4, GLYAT, GZMB, HLA-DQA1, IL2, INS, LEP, NCAM1, NOD2, SERPINA1, TIA1, TNF, TNFRSF8. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.