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Disease Info Card
Osteoarthropathy, Secondary Hypertrophic
Information about Osteoarthropathy, Secondary Hypertrophic: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Osteoarthropathy, Secondary Hypertrophic
Most recent studies have shown that Osteoarthropathy, Secondary Hypertrophic shares some biological mechanisms with adenocarcinoma, arthritis, bone-diseases, bronchogenic-carcinoma, carcinoma, clubbed-fingers, clubbing, hypertrophy, lung-diseases, lung-neoplasms, malignant-neoplasm-of-lung, malignant-neoplasms, malignant-squamous-cell-neoplasm, neoplasm-metastasis, neoplasms, osteoarthropathy, osteoarthropathy-primary-hypertrophic, pain, paraneoplastic-syndromes.
Among the many pathways, these few ones have gauged particular interests from scientists studying Osteoarthropathy, Secondary Hypertrophic, and have been seen in publications frequently: Acute-phase Response, Aging, Bone Remodeling, Bone Resorption, Cell Activation, Cell Adhesion, Cell Differentiation, Excretion, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Ossification, Pathogenesis, Platelet Aggregation, Reflex, Rumination, Secretion, Transport, Transposition
Quite a number of genes have been found to play important roles in Osteoarthropathy, Secondary Hypertrophic, such as ALB, AVP, BGLAP, CFTR, DBT, EGFR, GGH, GH1, GHRH, HGF, HMOX1, HPGD, HRAS, KNG1, MATN3, POMC, SLCO2A1, TNF, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.