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Disease Info Card
Osteoarthropathy, Primary Hypertrophic
Information about Osteoarthropathy, Primary Hypertrophic: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Osteoarthropathy, Primary Hypertrophic
Most recent studies have shown that Osteoarthropathy, Primary Hypertrophic shares some biological mechanisms with acromegaly, arthralgia, arthritis, arthropathy, bone-diseases, carcinoma, clubbed-fingers, clubbing, dermatologic-disorders, edema, hypertrophy, lung-neoplasms, neoplasms, osteoarthropathy, osteoarthropathy-secondary-hypertrophic, pachyderma, pain, periosteal-disorder, periostitis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Osteoarthropathy, Primary Hypertrophic, and have been seen in publications frequently: Aging, Bone Remodeling, Bone Resorption, Cell Activation, Cell Proliferation, Chemotaxis, Cytokine Production, Endocytosis, Excretion, Fibroblast Activation, Fibroblast Proliferation, Growth Hormone Secretion, Metaphase, Micropinocytosis, Ossification, Osteoblast Differentiation, Osteoclast Differentiation, Pathogenesis, Secretion, Transport
Quite a number of genes have been found to play important roles in Osteoarthropathy, Primary Hypertrophic, such as BGLAP, CBR1, DBT, DCN, FN1, HMOX1, HPGD, IGF1, IL1RN, IL6, JAK2, MATN3, PDP1, PPBP, SLCO2A1, TNF, TNFRSF11B, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.