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Disease Info Card
Oral Ulcer
Information about Oral Ulcer: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Oral Ulcer
Most recent studies have shown that Oral Ulcer shares some biological mechanisms with aphthous-stomatitis, arthritis, behcet-syndrome, edema, erythema, exanthema, hiv-infections, infective-disorder, inflammation, lupus-erythematosus-systemic, malignant-neoplasms, malignant-squamous-cell-neoplasm, mouth-diseases, mouth-neoplasms, neoplasms, pain, stomatitis, tongue-diseases, ulcer, vasculitis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Oral Ulcer, and have been seen in publications frequently: Aging, Angiogenesis, Cell Adhesion, Cell Cycle, Cell Proliferation, Chemotaxis, Coagulation, Dehiscence, Fibrinolysis, Glomerular Filtration, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Pathogenesis, Pigmentation, Secretion, Sensitization, Transport, Wound Healing
Quite a number of genes have been found to play important roles in Oral Ulcer, such as ACR, C3, C4A, CABIN1, CALR, CD4, CRP, CSF2, CSF3, EIF3M, ESR1, FGF7, HLA-B, HLA-DRB4, IL6, MTOR, SRL, STT3A, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.