Disease Info Card

Ocular Myasthenia

Information about Ocular Myasthenia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Ocular Myasthenia

Most recent studies have shown that Ocular Myasthenia shares some biological mechanisms with autoimmune-diseases, autoimmune-reaction, blepharoptosis, diplopia, disorder-of-eye, generalized-myasthenia, muscle-weakness, myasthenia-gravis, myasthenia-gravis-generalized, myasthenias, myopathy, neoplasms, neuromuscular-diseases, ocular-motility-disorders, ophthalmoplegia, strabismus, thymoma, thymus-neoplasms, weakness, widespread-disease.

Among the many pathways, these few ones have gauged particular interests from scientists studying Ocular Myasthenia, and have been seen in publications frequently: Aging, Cell Recognition, Complement Activation, Dehiscence, Immune Response, Innervation, Lipid Storage, Localization, Mastication, Muscle Atrophy, Pathogenesis, Reflex, Regeneration, Secretion, Sensitization

Quite a number of genes have been found to play important roles in Ocular Myasthenia, such as ACHE, AR, BCHE, C3, CASP1, CD4, CD55, CES2, DMPK, HLA-A, HLA-B, IL18, MUSK, OMG, POMC, TNFRSF25. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Ocular Myasthenia Related Genes

click to see detail information for each gene

ACHE AR BCHE
C3 CASP1 CD4
CD55 CES2 DMPK
HLA-A HLA-B IL18
MUSK OMG POMC
TNFRSF25

Pathways Related to Ocular Myasthenia

This information is being compiled and will come in a future update

Aging Cell Recognition Complement Activation
Dehiscence Immune Response Innervation
Lipid Storage Localization Mastication
Muscle Atrophy Pathogenesis Reflex
Regeneration Secretion Sensitization