Disease Info Card

Myasthenia Gravis, Generalized

Information about Myasthenia Gravis, Generalized: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Myasthenia Gravis, Generalized

Most recent studies have shown that Myasthenia Gravis, Generalized shares some biological mechanisms with autoimmune-diseases, autoimmune-reaction, blepharoptosis, decreased-immunologic-activity-[pe], deglutition-disorders, diplopia, generalized-myasthenia, hyperplasia, muscle-weakness, myasthenia-gravis, myasthenia-gravis-crisis, myasthenias, neoplasms, neuromuscular-diseases, ocular-motility-disorders, ocular-myasthenia, thymoma, thymus-hyperplasia, thymus-neoplasms, weakness.

Among the many pathways, these few ones have gauged particular interests from scientists studying Myasthenia Gravis, Generalized, and have been seen in publications frequently: B Cell Proliferation, Cell Proliferation, Cell Recognition, Complement Activation, Dehiscence, Hypersensitivity, Immune Response, Innervation, Localization, Muscle Contraction, Pathogenesis, Response To Tacrolimus, Sensitization, Skeletal Muscle Contraction, Synaptic Transmission, Visual Learning

Quite a number of genes have been found to play important roles in Myasthenia Gravis, Generalized, such as ACHE, AGRN, ALB, BCHE, C3, CD4, CP, EIF3K, IL2, KRT20, MS4A1, MUSK, OMG, POMC, RAPSN, TTN. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.