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Disease Info Card
Nephrolithiasis, Calcium Oxalate
Information about Nephrolithiasis, Calcium Oxalate: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Nephrolithiasis, Calcium Oxalate
Most recent studies have shown that Nephrolithiasis, Calcium Oxalate shares some biological mechanisms with albinism, cat-diseases, dog-diseases, hypercalcemia, hypercalciuria, hyperoxalaemia, hyperoxaluria, hyperoxaluria-primary, injury-to-kidney, kidney-calculi, kidney-failure, lithiasis, metabolic-diseases, nephrocalcinosis, nephrolithiasis, recurrence-(disease-attribute), tissue-adhesions, urolithiasis, urologic-diseases.
Among the many pathways, these few ones have gauged particular interests from scientists studying Nephrolithiasis, Calcium Oxalate, and have been seen in publications frequently: Blood Coagulation, Circadian Rhythm, Coagulation, Diuresis, Endocytosis, Energy Homeostasis, Excretion, Fibrinolysis, Glomerular Filtration, Intestinal Absorption, Ion Transport, Localization, Menopause, Oxalate Transport, Pathogenesis, Protein Secretion, Regeneration, Reverse Transcription, Secretion, Transport
Quite a number of genes have been found to play important roles in Nephrolithiasis, Calcium Oxalate, such as AGXT, AMBP, CAT, CRAT, CXXC1, F2, GGCX, GGT1, GGTLC1, GLYAT, HOGA1, PODXL2, PTH, SAT1, SLC13A1, SLC26A1, SLC26A6, SLC38A1, SPP1, UMOD. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.