Disease Info Card

Neonatal Torulopsis Glabrata Fungemia

Information about Neonatal Torulopsis Glabrata Fungemia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Neonatal Torulopsis Glabrata Fungemia

Most recent studies have shown that Neonatal Torulopsis Glabrata Fungemia shares some biological mechanisms with abscess, aspergillosis, candidiasis, candidiasis-vulvovaginal, cross-infection, diabetes-mellitus, endocarditis, fungemia, infection-by-cryptococcus-neoformans, infective-disorder, leukemia, lung-diseases-fungal, malignant-neoplasms, mycoses, neoplasms, opportunistic-infections, oral-candidiasis, pneumonia, systemic-infection, urinary-tract-infection.

Among the many pathways, these few ones have gauged particular interests from scientists studying Neonatal Torulopsis Glabrata Fungemia, and have been seen in publications frequently: Cell Division, Cell Growth, Drug Resistance, Electron Transport, Electron Transport Chain, Fermentation, Killer Activity, Localization, Mitochondrial Translation, Nadh Oxidation, Oxidative Phosphorylation, Pathogenesis, Phagocytosis, Pigmentation, Regeneration, Respiratory Burst, Secretion, Translation, Transport, Virulence

Quite a number of genes have been found to play important roles in Neonatal Torulopsis Glabrata Fungemia, such as AKR1A1, AVP, C12ORF57, C2, CALB2, CD55, CNOT8, CR2, DHRS4, FANCD2, PLXNA1, QPCT, RPL10, TDGF1, TNF, TNFRSF10B. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.