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Disease Info Card
Endocarditis
Information about Endocarditis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Endocarditis
Most recent studies have shown that Endocarditis shares some biological mechanisms with abscess, aneurysm, aortic-valve-insufficiency, bacteremia, bacterial-endocarditis, embolism, heart-diseases, heart-failure, heart-valve-disease, hemorrhage, infective-disorder, infective-endocarditis, mitral-valve-insufficiency, regurgitation, rheumatism, staphylococcal-infections, streptococcal-infections, subacute-bacterial-endocarditis, systemic-infection.
Among the many pathways, these few ones have gauged particular interests from scientists studying Endocarditis, and have been seen in publications frequently: Aging, Biofilm Formation, Cardiac Conduction, Coagulation, Dehiscence, Excretion, Fermentation, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Pathogenesis, Phagocytosis, Platelet Activation, Platelet Aggregation, Response To Antibiotic, Secretion, Transport, Transposition, Virulence
Quite a number of genes have been found to play important roles in Endocarditis, such as ALB, C3, CAT, CPB1, CRAT, CRP, CSRP1, F3, FN1, GLYAT, GNL3, IL6, KRAS, MVP, SLC9A6, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.