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Disease Info Card
Lichen Sclerosus Et Atrophicus
Information about Lichen Sclerosus Et Atrophicus: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Lichen Sclerosus Et Atrophicus
Most recent studies have shown that Lichen Sclerosus Et Atrophicus shares some biological mechanisms with atrophy, carcinoma, dermatologic-disorders, hyperplasia, intraepithelial-neoplasia, lichen-disease, lichen-planus, localized-scleroderma, malignant-neoplasm-of-vulva, malignant-neoplasms, malignant-squamous-cell-neoplasm, morphea, neoplasms, precancerous-conditions, pruritus, sclerosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Lichen Sclerosus Et Atrophicus, and have been seen in publications frequently: Aging, Angiogenesis, Cell Cycle, Cell Proliferation, Cytokine Production, Defecation, Exocytosis, Immune Response, Inflammatory Response, Keratinization, Localization, Menarche, Menopause, Methylation, Oncogenesis, Pathogenesis, Pigmentation, Regeneration, Senescence, Sensitization
Quite a number of genes have been found to play important roles in Lichen Sclerosus Et Atrophicus, such as ATP6V0A1, CD4, CD44, CD8A, CDKN2A, ECM1, ELN, IL1RN, ITCH, LIAS, MBP, RFC1, RFC2, RFC4, SERPINB3, TP53, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.