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Disease Info Card
Leprosy, Paucibacillary
Information about Leprosy, Paucibacillary: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Leprosy, Paucibacillary
Most recent studies have shown that Leprosy, Paucibacillary shares some biological mechanisms with borderline-lepromatous-leprosy, borderline-leprosy, borderline-tuberculoid-leprosy, communicable-diseases, delayed-hypersensitivity, erythema, granuloma, hiv-infections, indeterminate-leprosy, inflammation, leprosy, leprosy-lepromatous, leprosy-multibacillary, nerve-damage, neuritis, tuberculoid-leprosy, tuberculosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Leprosy, Paucibacillary, and have been seen in publications frequently: Angiogenesis, B Cell Activation, Cell Activation, Complement Activation, Drug Resistance, Granuloma Formation, Humoral Immune Response, Hypersensitivity, Immune Response, Inflammatory Response, Leukocyte Migration, Localization, Macrophage Activation, Pathogenesis, Phagocytosis, Protein Secretion, Reflex, Regeneration, Response To Peptide, Secretion
Quite a number of genes have been found to play important roles in Leprosy, Paucibacillary, such as BTN1A1, C3, CACNA1A, CRP, HLA-DRB4, HPD, HTN3, IFNG, IL10, IL2, MB, NOD2, SLC11A1, SLC4A7, SPINT2, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.