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Disease Info Card
Hypotrichosis
Information about Hypotrichosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hypotrichosis
Most recent studies have shown that Hypotrichosis shares some biological mechanisms with atrophic-condition-of-skin, atrophy, basal-cell-carcinoma, christ-siemens-touraine-syndrome, congenital-hypotrichia, dermatologic-disorders, developmental-absence-of-tooth, dwarfism, dysplasia, dystrophy, ectodermal-dysplasia, hair-diseases, hypodontia, hypohidrosis, hypoplasia, hypotrichosis-simplex, ichthyoses, palmoplantar-keratosis, skin-neoplasms, tooth-abnormalities.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hypotrichosis, and have been seen in publications frequently: Aging, Anagen, Catagen, Cell Adhesion, Cell Differentiation, Cell Proliferation, Cell-cell Adhesion, Cornification, Excretion, Hair Cycle, Hair Follicle Development, Hair Follicle Morphogenesis, Keratinization, Localization, Ossification, Pathogenesis, Photoprotection, Pigmentation, Telogen, Translation
Quite a number of genes have been found to play important roles in Hypotrichosis, such as APCDD1, CDH15, CDH3, CDSN, DSC3, DSG1, DSG4, EDA, GJA1, HR, LIPC, LIPH, LPAR4, LPAR6, PKP1, SOX18, ST14. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.