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- Table of Contents
Information about Hypoglycemia Of Infancy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Hypoglycemia Of Infancy shares some biological mechanisms with adenomatous-hyperplasia, brain-injuries, diabetes-mellitus, diabetes-mellitus-insulin-dependent, diabetes-mellitus-non-insulin-dependent, focal-glandular-hyperplasia, hereditary-diseases, hyperinsulinism, hyperplasia, hypoglycemia, insulinoma, neonatal-hypoglycemia, neoplasms, nesidioblastosis, pancreatic-diseases, pancreatic-neoplasm, persistent-hyperinsulinemia-hypoglycemia-of-infancy, sequelae-aspects.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hypoglycemia Of Infancy, and have been seen in publications frequently: Cell Cycle, Cell Differentiation, Cell Division, Cell Motility, Cell Proliferation, Enucleation, Exocytosis, Glucose Homeostasis, Glycosylation, Hormone Secretion, Insulin Secretion, Lipid Homeostasis, Localization, Pathogenesis, Regulation Of Insulin Secretion, Response To Camp, Secretion, Transepithelial Transport, Transport, Tricarboxylic Acid Cycle
Quite a number of genes have been found to play important roles in Hypoglycemia Of Infancy, such as ABCB6, ABCC1, ABCC8, CFTR, FOXA2, GCG, GCK, GK, GLUD1, HADH, IGF2, INS, KCNJ11, MAP4K2, PDX1, POMC, SST. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.