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Disease Info Card
Hyperargininemia
Information about Hyperargininemia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hyperargininemia
Most recent studies have shown that Hyperargininemia shares some biological mechanisms with argininosuccinic-aciduria, atrophy, brain-injuries, citrullinemia, cystinuria, encephalopathies, epilepsy, growth-retardation, hyperammonemia, inborn-errors-of-metabolism, liver-cirrhosis, metabolic-diseases, muscle-spasticity, ornithine-carbamoyltransferase-deficiency, phenylketonurias, urea-cycle-disorders-inborn.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hyperargininemia, and have been seen in publications frequently: Arginine Homeostasis, Arginine Transport, Cell Cycle, Drug Resistance, Energy Homeostasis, Excretion, Immune Response, Inflammatory Response, Localization, Menstruation, Myelination, Ovulation, Parturition, Pathogenesis, Proteolysis, Sensitization, Transport, Tryptophan Transport, Urea Cycle, Wound Healing
Quite a number of genes have been found to play important roles in Hyperargininemia, such as ACADS, ARG1, ARG2, ASL, ASS1, ATP6V0A1, CSF2, LAMC2, NAGS, NOS1, NOS2, OTC, RFC1, RFC2, RFC4, TINAGL1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.