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Disease Info Card
Greig Cephalopolysyndactyly Syndrome
Information about Greig Cephalopolysyndactyly Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Greig Cephalopolysyndactyly Syndrome
Most recent studies have shown that Greig Cephalopolysyndactyly Syndrome shares some biological mechanisms with acrocallosal-syndrome, cell-transformation-neoplastic, cerebellar-neoplasms, chromosomal-translocation, chronic-pain, craniofacial-abnormalities, craniofacial-dysostosis, depressive-disorder, developmental-delay-(disorder), haploinsufficiency, malignant-neoplasms, medulloblastoma, neoplasms, orbital-separation-excessive, pain, pallister-hall-syndrome, polydactyly, polysyndactyly, syndactyly.
Among the many pathways, these few ones have gauged particular interests from scientists studying Greig Cephalopolysyndactyly Syndrome, and have been seen in publications frequently: Cell Cycle, Cell Migration, Cell Proliferation, Cellular Localization, Cerebellar Granule Cell Precursor Proliferation, Cerebellum Development, Dedifferentiation, Exocytosis, Interphase, Limb Development, Localization, Microtubule Nucleation, Microtubule Polymerization, Neurogenesis, Ossification, Pathogenesis, Protein Phosphorylation, Regeneration, Stomatal Closure, Stomatal Movement
Quite a number of genes have been found to play important roles in Greig Cephalopolysyndactyly Syndrome, such as BTG2, CDKN1A, CDKN1B, GAP43, GCK, GLI1, GLI3, GOLGB1, OPN1MW, PCBD1, PTCH1, SHH, SNAP25, SUB1, TNFSF14, TUBGCP2, TUBGCP3, TUBGCP4. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.