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Disease Info Card
Fibrodysplasia Ossificans Progressiva
Information about Fibrodysplasia Ossificans Progressiva: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Fibrodysplasia Ossificans Progressiva
Most recent studies have shown that Fibrodysplasia Ossificans Progressiva shares some biological mechanisms with ankylosis, calcinosis, congenital-abnormality, congenital-disorders, connective-tissue-diseases, dysplasia, edema, hereditary-diseases, heterotopic-ossification, inflammation, myositis, myositis-ossificans, neoplasms, pain, pseudohypoparathyroidism, temporomandibular-joint-disorders.
Among the many pathways, these few ones have gauged particular interests from scientists studying Fibrodysplasia Ossificans Progressiva, and have been seen in publications frequently: Aging, Angiogenesis, Bmp Signaling Pathway, Bone Remodeling, Bone Resorption, Cardiac Conduction, Endochondral Ossification, Excretion, Inflammatory Response, Limb Development, Localization, Ossification, Osteoblast Differentiation, Pathogenesis, Protein Phosphorylation, Regeneration, Segmentation, Tissue Development, Tissue Homeostasis, Transport
Quite a number of genes have been found to play important roles in Fibrodysplasia Ossificans Progressiva, such as ACVR1, BMP2, BMP4, CAT, CHTOP, CRAT, FGFR1OP, FKBP1A, GLUL, GLYAT, GNAS, MTSS1, NOG, POMC, RUNX2, SMAD1, SMAD5. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.