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Disease Info Card
Farber Lipogranulomatosis
Information about Farber Lipogranulomatosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Farber Lipogranulomatosis
Most recent studies have shown that Farber Lipogranulomatosis shares some biological mechanisms with arthropathy, edema, erdheim-chester-disease, gaucher-disease, granuloma, hoarseness, inborn-errors-of-metabolism, leukodystrophy, leukodystrophy-metachromatic, lipoidosis, lysosomal-storage-diseases, nervousness, niemann-pick-disease-type-c, niemann-pick-diseases, pigmentation-disorders, respiratory-distress-syndrome-newborn, sphingolipidoses, storage-disease.
Among the many pathways, these few ones have gauged particular interests from scientists studying Farber Lipogranulomatosis, and have been seen in publications frequently: Brain Development, Cell Death, Cell Differentiation, Cell Growth, Cell Proliferation, Coagulation, Endocytosis, Excretion, Genetic Transfer, Glycosylation, Granuloma Formation, Induction Of Apoptosis, Intracellular Transport, Lipid Storage, Localization, Pathogenesis, Pigmentation, Proteolysis, Receptor-mediated Endocytosis, Transport
Quite a number of genes have been found to play important roles in Farber Lipogranulomatosis, such as ACER1, ACER2, ARSA, ASAH1, CASP3, CCL5, FAS, KLF6, MAPK1, MAPK3, MBTPS1, MTCH1, NAGA, PAEP, PSAP, QPCT, SMPD1, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.