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Disease Info Card
Familial Cold Urticaria
Information about Familial Cold Urticaria: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Familial Cold Urticaria
Most recent studies have shown that Familial Cold Urticaria shares some biological mechanisms with amyloidosis, arthralgia, arthritis, autoimmune-diseases, brucellosis, chronic-infantile-neurological-cutaneous-and-articular-syndrome, conjunctivitis, cryopyrin-associated-periodic-syndromes, exanthema, familial-mediterranean-fever, hereditary-autoinflammatory-diseases, hyper-igd-syndrome, inflammation, inflammatory-disorder, muckle-wells-syndrome, sensorineural-hearing-loss-(disorder), urticaria, urticaria-due-to-cold.
Among the many pathways, these few ones have gauged particular interests from scientists studying Familial Cold Urticaria, and have been seen in publications frequently: Acute-phase Response, Aging, Cell Activation, Cell Cycle, Cell Death, Chromosome Condensation, Cognition, Cytokine Secretion, Dna Replication, Hypersensitivity, Immune Response, Inflammatory Response, Innate Immune Response, Interleukin-1 Production, Mast Cell Activation, Pathogenesis, Response To Cold, Secretion, Translation, Tube Formation
Quite a number of genes have been found to play important roles in Familial Cold Urticaria, such as CADPS, CAPS, CASP1, CRP, IL18, IL1B, IL1RN, IL6, LYZ, MEFV, MVK, NFKB1, NLRP3, NOD2, PYCARD, STS, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.