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Disease Info Card
Extrinsic Allergic Alveolitis
Information about Extrinsic Allergic Alveolitis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Extrinsic Allergic Alveolitis
Most recent studies have shown that Extrinsic Allergic Alveolitis shares some biological mechanisms with allergy, asthma, bird-fanciers-lung, bronchitis, coughing, dyspnea, farmers-lung, fibrosis, granuloma, inflammation, lung-diseases, lung-diseases-interstitial, pneumonia, pneumonia-interstitial, pulmonary-fibrosis, respiratory-hypersensitivity, sarcoidosis, usual-interstitial-pneumonitis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Extrinsic Allergic Alveolitis, and have been seen in publications frequently: Anaphylaxis, Angiogenesis, Cell Activation, Complement Activation, Cytokine Production, Exogen, Granuloma Formation, Humoral Immune Response, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Lymphocyte Proliferation, Macrophage Activation, Pathogenesis, Phagocytosis, Secretion, Sensitization, T Cell Activation, Transport
Quite a number of genes have been found to play important roles in Extrinsic Allergic Alveolitis, such as ACE, ALB, CD4, CD8A, CTLA4, DLC1, HLA-DQA1, HP, IFNG, IL10, IL13, IL2, IL4, IL6, MUC1, NOD2, TNF, TUB. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.