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- Table of Contents
Information about Extraskeletal Myxoid Chondrosarcoma: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Extraskeletal Myxoid Chondrosarcoma shares some biological mechanisms with bone-neoplasms, carcinoma, cardiovirus-infections, chondrosarcoma, cryoglobulinemia, diabetes-mellitus, enterovirus-infections, essential-mixed-cryoglobulinemia, hepatitis, malignant-neoplasms, malignant-paraganglionic-neoplasm, mixed-cryoglobulinaemia, myocarditis, neoplasm-metastasis, neoplasms, sarcoma, soft-tissue-neoplasms, virus-diseases.
Among the many pathways, these few ones have gauged particular interests from scientists studying Extraskeletal Myxoid Chondrosarcoma, and have been seen in publications frequently: Cell Adhesion, Cell Cycle, Cell Death, Cell Differentiation, Cell Growth, Cell Proliferation, Immune Response, Localization, Organ Development, Pathogenesis, Proteolysis, Reflex, Secretion, Sensory Organ Development, Translation, Transport, Tropism, Viral Replication, Wound Healing
Quite a number of genes have been found to play important roles in Extraskeletal Myxoid Chondrosarcoma, such as ALB, CKAP4, ETFA, EWSR1, FN1, FUS, IFNA1, INS, MUC1, NR4A3, RASIP1, RPE65, S100A1, S100B, TAF15, TEC, TNF, TP63, UVRAG, VIM. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.