Disease Info Card

Epiphysis Disorders

Information about Epiphysis Disorders: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Epiphysis Disorders

Most recent studies have shown that Epiphysis Disorders shares some biological mechanisms with arthritis, avascular-necrosis-of-bone, bone-necrosis, bone-neoplasms, chondrolysis, congenital-abnormality, degenerative-polyarthritis, dislocations, dysplasia, epiphyses-slipped, femur-head-necrosis, fracture, hip-dislocation-congenital, hypertrophy, legg-calve-perthes-disease, neoplasms, pain, slipped-capital-femoral-epiphyses, slipped-upper-femoral-epiphysis, tibial-fractures.

Among the many pathways, these few ones have gauged particular interests from scientists studying Epiphysis Disorders, and have been seen in publications frequently: Aging, Angiogenesis, Bone Development, Bone Maturation, Bone Remodeling, Bone Resorption, Cell Proliferation, Chondrocyte Differentiation, Chondrocyte Hypertrophy, Circadian Rhythm, Endochondral Ossification, Excretion, Innervation, Localization, Ossification, Pathogenesis, Regeneration, Secretion, Translation, Transport

Quite a number of genes have been found to play important roles in Epiphysis Disorders, such as BEST1, BGLAP, DMD, GGH, GH1, GPSM2, HHIP, IGF1, PTH, PTRH1, REG3A, RPL29, SS18L1, ST13, TNFSF14, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.