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Disease Info Card
Endometrial Hyperplasia
Information about Endometrial Hyperplasia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Endometrial Hyperplasia
Most recent studies have shown that Endometrial Hyperplasia shares some biological mechanisms with adenocarcinoma, atypical-endometrial-hyperplasia, carcinoma, endometrial-adenocarcinoma, endometrial-carcinoma, endometrial-neoplasms, endometrial-polyp, endometriosis-site-unspecified, hemorrhage, hyperplasia, malignant-neoplasms, malignant-paraganglionic-neoplasm, neoplasms, polyps, precancerous-conditions, uterine-diseases, uterine-fibroids, uterine-hemorrhage, uterine-neoplasms.
Among the many pathways, these few ones have gauged particular interests from scientists studying Endometrial Hyperplasia, and have been seen in publications frequently: Aging, Angiogenesis, Cell Cycle, Cell Differentiation, Cell Growth, Cell Proliferation, Coagulation, Estrus, Excretion, Fertilization, Localization, Menarche, Menopause, Menstruation, Methylation, Mismatch Repair, Mitosis, Ovulation, Pathogenesis, Secretion
Quite a number of genes have been found to play important roles in Endometrial Hyperplasia, such as ASAP1, ASAP2, BCL2, BRD2, CYP19A1, DBT, ESR1, MLH1, MRPS30, PAPOLA, PCNA, PGR, PLOD1, PTEN, REG3A, TMEM37, TP53. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.